Apoptosis: How to increase cell life.

There are two ways that a cell can die: necrosis and apoptosis as cells begin to die you begin to die slowly. There is various way to slow down and prevent this process. You prolong cell life with a sentient, alkaline, and yogic diet.

1. Necrosis occurs when a cell is damaged by an external force, such as poison, a bodily injury, infection, or getting cut off from the blood supply (which might occur during a heart attack or stroke). When cells die from necrosis, it’s a rather messy affair. Death causes inflammation that can cause further distress or injury within the body.
2. Apoptosis, on the other hand, is relatively civil, even though it may not sound so at first — it’s when a cell commits suicide. How is that better than necrosis? For one thing, the cleanup is much easier. It’s sometimes referred to as programmed cell death, and indeed, the process of apoptosis follows a controlled, predictable routine.

When a cell is compelled to commit suicide (we’ll get to the triggers for apoptosis in just a minute), proteins called caspases go into action. They break down the cellular components needed for survival, and they spur the production of enzymes known as DNases, which destroy the DNA in the nucleus of the cell. It’s like roadies breaking down the stage in an arena after a major band has been through town. The cell shrinks and sends out distress signals, which are answered by vacuum cleaners known as macrophages. The macrophages clean away the shrunken cells, leaving no trace, so these cells have no chance of causing the damage that necrotic cells do.

Apoptosis also differs from necrosis in that it’s essential to human development. For example, in the womb, our fingers and toes are connected by a sort of webbing. Apoptosis is what causes that webbing to disappear, leaving us with ten separate digits. As our brains develop, the body creates millions of more cells than it needs; the ones that don’t form synaptic connections undergo apoptosis so that the remaining cells function well. Programmed cell death is also necessary to start the process of menstruation.

That’s not to say that apoptosis is a perfect process. Sometimes, the wrong cells kill themselves off, and sometimes, the ones that should say “Auf Wiedersehen” stick around instead. This brings us to our discussion of the triggers of apoptosis. Rather than dying due to injury, cells that go through apoptosis die in response to signals within the body. When cells recognize viruses and gene mutations, they may induce death to prevent the damage from spreading. When cells are under stress, as may happen when free radicals are on the loose or when a person undergoes radiation, apoptosis can occur. But there are also signals within the body that send the message that a cell should continue living. All cells have varying levels of sensitivity to the positive and negative triggers, so sometimes the wrong cells live and die.

Scientists are trying to learn how they can modulate apoptosis so that they can control which cells live and which undergo programmed cell death. Anti-cancer drugs and radiation, for example, work by triggering apoptosis in diseased cells. Many diseases and disorders are linked with the life and death of cells — increased apoptosis is a characteristic of AIDS, Alzheimer’s, and Parkinson’s disease, while decreased apoptosis can signal lupus or cancer. Understanding how to regulate apoptosis could be the first step to treating these conditions.

Solutions are in your diet: Consume Plant-based food, Avoid all kinds of processed food, remove toxic food from your diet adopt a sentient diet.

Lemons and lemon oil are popular because of their refreshing scent and invigorating, purifying, and cleansing properties. Research shows that lemon oil contains powerful antioxidants and helps to reduce inflammation, fight bacteria and fungi, boost energy levels and ease digestion.

Chronic inflammation causes major diseases like All kinds of Heart issues, Cancer, Autism, Obesity, Diabetes, Dementia, Depression, Different kinds of syndromes, Autoimmune deficiency (Aids)

Studies show that a particular essential oil may provide a solid defense against various cancers. Lemongrass essential oil, which contains high levels of an aromatic terpene known as linalool, has been used traditionally in tropical countries in the treatment of various health conditions. What’s even more exciting is that some of the latest in vitro and in vivo animal studies suggest that it could be at least as effective as chemotherapy in targeting cancer cells – without the harmful toxic effects of chemotherapy.

Long prized for its aroma-therapeutic qualities, Cymbopogon citratus (lemongrass or lemongrass) is loaded with key nutrient compounds that present powerful antibacterial, anti-fungal, anti-inflammatory, and anticancer potential. A 2011 study published in the Journal of Advanced Pharmaceutical Technology & Research outlines the dozens of unique phytoconstituents present in lemongrass, including a diverse array of terpenes, ketones, esters, flavonoids, and phenolic compounds.

The Long List of Health Benefits of Lemongrass Essential Oil

The paper also highlights the many scientifically-confirmed benefits of lemongrass, which are that it:

• Destroys harmful pathogens
• Promotes healthy stools
• Inhibits damaging fungi
• Reduces inflammation
• Protects against DNA damage
• Guards against malaria
• Neutralizes damaging free radicals
• Helps to balance cholesterol levels
• Modulates healthy sleep patterns
• And so much more

According to the report authors…

Studies indicate that Cymbopogon citratus (a variety of lemongrass) possesses various pharmacological activities such as anti-amoebic, antibacterial, antidiarrheal, antifilarial, antifungal, and anti-inflammatory properties. Various other effects such as antimalarial, antimutagenicity, antimycobacterial, antioxidants, hypoglycemic, and neurobehavioral have also been studied.

The Unbelievable Anti-Cancer Effects of Lemongrass Essential Oil

https://science.howstuffworks.com/life/cellular-microscopic/apoptosis.htm